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One possibility would be combining GHK with antioxidants, such as the mitochondrial targeted antioxidant SS31 [23], given the role of redox imbalance in persistent lung fibrosis [15]

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FIGURE 1 Patient-Related Factors The study of why and how ADAs are generated is complicated by the fact that some patients develop ADAs and some, with the same clinical indication and receiving the same therapeutic mAb, do not

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