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demonstrated that the addition of TGF-β1 monoclonal antibody to renin-angiotensin system (RAS) inhibitors did not slow the progression of diabetic nephropathy, suggesting that TGF-β signaling is not exclusively the main driving force in CKD progression [3]
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(2008) found that Semax could protect neurons from oxidative stress and apoptosis, suggesting potential applications in the treatment of neurodegenerative diseases such as Alzheimer's and Parkinson's disease
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