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Following this model, standard therapies often fail to eliminate CSCs, as they primarily target rapidly proliferating and differentiated cancer cells rather than the slow-cycling, therapy-resistant stem cell-like population

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The authors assumed that when a higher concentration of intravenous NAC is given, better clinical outcomes could be expected because of the effective reduction in viral replication and significant alleviation of pneumocyte damage, as well as the modulation of immune responses and therefore prevention of a “cytokine storm” [133]