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As TBI remains to be a major cause of morbidity and mortality worldwide, there is increasing demand on enhancing our collective understanding of the underlying pathophysiology and chronic neuropsychiatric sequelae post TBI

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Considering the fact that Baf-A1 suppresses fusion of autophagosomes with the lysosomes 39 , our finding that DFO protected against ferroptosis suggests that lysosomal iron pool is involved in GSH depletion-induced cell death 48